SP600125, an anthrapyrazolone inhibitor of Jun N-terminal kinase | PNAS
SP600125 | CAS 129-56-6 | SCBT - Santa Cruz Biotechnology
SP600125 enhances C-2-induced cell death by the switch from autophagy to apoptosis in bladder cancer cells | Journal of Experimental & Clinical Cancer Research | Full Text
SP600125 enhances C-2-induced cell death by the switch from autophagy to apoptosis in bladder cancer cells | Journal of Experimental & Clinical Cancer Research | Full Text
JNK inhibitor SP600125 promotes the formation of polymerized tubulin, leading to G2/M phase arrest, endoreduplication, and delayed apoptosis | Experimental & Molecular Medicine
Effect of specific JNK inhibitor SP600125 (SP) on the effect of 1 mM... | Download Scientific Diagram
JNK inhibitor SP600125 promotes the formation of polymerized tubulin, leading to G2/M phase arrest, endoreduplication, and delayed apoptosis | Experimental & Molecular Medicine
JNK inhibitor SP600125 protects against lipopolysaccharide‑induced acute lung injury via upregulation of claudin‑4
JNK inhibitor SP600125 promotes the formation of polymerized tubulin, leading to G2/M phase arrest, endoreduplication, and delayed apoptosis | Experimental & Molecular Medicine
A c-Jun NH2-Terminal Kinase Inhibitor SP600125 (Anthra[1,9-cd]pyrazole-6 (2H)-one) Blocks Activation of Pancreatic Stellate Cells | Journal of Pharmacology and Experimental Therapeutics
IJMS | Free Full-Text | SP600125 Induces Src and Type I IGF Receptor Phosphorylation Independent of JNK
The JNK inhibitor SP600125 enhances dihydroartemisinin-induced apoptosis by accelerating Bax translocation into mitochondria in human lung adenocarcinoma cells - ScienceDirect